Editorial: Bidirectional Communication Between Synapses and Nucleus in Brain Physiology and Disease

Arnaldo Parra-Damas; Toh Hean Ch'ng; Bryen A. Jordan; Carlos A. Saura

doi:10.3389/fnmol.2022.909036

Editorial: Bidirectional Communication Between Synapses and Nucleus in Brain Physiology and Disease

Arnaldo Parra-Damas^*, Toh Hean Ch'ng, Bryen A. Jordan, Carlos A. Saura

^*Corresponding author for this work

Research output: Contribution to journal › Review article › Research › peer-review

Original language	English
Article number	909036
Journal	Frontiers in Molecular Neuroscience
Volume	15
DOIs	https://doi.org/10.3389/fnmol.2022.909036
Publication status	Published - 6 May 2022

Keywords

local translation
neurodegeneration
neurodevelopmental
neuropsychiatric
synapse-to-nucleus
synaptonuclear
transcription factors

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10.3389/fnmol.2022.909036

https://ddd.uab.cat/record/293090

Cite this

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title = "Editorial: Bidirectional Communication Between Synapses and Nucleus in Brain Physiology and Disease",

keywords = "local translation, neurodegeneration, neurodevelopmental, neuropsychiatric, synapse-to-nucleus, synaptonuclear, transcription factors",

author = "Arnaldo Parra-Damas and Ch'ng, {Toh Hean} and Jordan, {Bryen A.} and Saura, {Carlos A.}",

note = "Funding Information: CAS was supported by grants from the Ministerio de Ciencia e Innovaci{\'o}n (FEDER funds) PID2019-106615RB-I00, Instituto de Salud Carlos III (CIBERNED CB06/05/0042), and Generalitat de Catalunya (2017 SGR749). AP-D was supported by a Juan de la Cierva-Incorporaci{\'o}n Program fellowship (IJC2019-042468-I). THC was supported by Nanyang Assistant Professor (NAP) startup funds from Nanyang Technological University, Singapore and by Singapore Ministry of Education Academic Research Fund Tier 1 (MOE2018-T1-002-033). BAJ was supported by National Institutes of Health (R01NS118820). The funders had no role in the design, data collection and analysis or preparation of the manuscript. ",

year = "2022",

month = may,

day = "6",

doi = "10.3389/fnmol.2022.909036",

language = "English",

volume = "15",

journal = "Frontiers in Molecular Neuroscience",

issn = "1662-5099",

publisher = "Frontiers Media S.A.",

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T1 - Editorial

T2 - Bidirectional Communication Between Synapses and Nucleus in Brain Physiology and Disease

AU - Parra-Damas, Arnaldo

AU - Ch'ng, Toh Hean

AU - Jordan, Bryen A.

AU - Saura, Carlos A.

N1 - Funding Information: CAS was supported by grants from the Ministerio de Ciencia e Innovación (FEDER funds) PID2019-106615RB-I00, Instituto de Salud Carlos III (CIBERNED CB06/05/0042), and Generalitat de Catalunya (2017 SGR749). AP-D was supported by a Juan de la Cierva-Incorporación Program fellowship (IJC2019-042468-I). THC was supported by Nanyang Assistant Professor (NAP) startup funds from Nanyang Technological University, Singapore and by Singapore Ministry of Education Academic Research Fund Tier 1 (MOE2018-T1-002-033). BAJ was supported by National Institutes of Health (R01NS118820). The funders had no role in the design, data collection and analysis or preparation of the manuscript.

PY - 2022/5/6

Y1 - 2022/5/6

KW - local translation

KW - neurodegeneration

KW - neurodevelopmental

KW - neuropsychiatric

KW - synapse-to-nucleus

KW - synaptonuclear

KW - transcription factors

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U2 - 10.3389/fnmol.2022.909036

DO - 10.3389/fnmol.2022.909036

M3 - Review article

C2 - 35600080

AN - SCOPUS:85130732726

SN - 1662-5099

VL - 15

JO - Frontiers in Molecular Neuroscience

JF - Frontiers in Molecular Neuroscience

M1 - 909036

ER -

Editorial: Bidirectional Communication Between Synapses and Nucleus in Brain Physiology and Disease

Keywords

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Modulación terapéutica del epitranscriptoma neuronal en demencia

SYNAPTIC INTERACTOME AND TRANSCRIPTOME OF THE SYNAPTONUCLEAR FACTOR CRTC1 IN ALZHEIMER DISEASE TRANSGENIC MICE

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Editorial: Bidirectional Communication Between Synapses and Nucleus in Brain Physiology and Disease

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Modulación terapéutica del epitranscriptoma neuronal en demencia

SYNAPTIC INTERACTOME AND TRANSCRIPTOME OF THE SYNAPTONUCLEAR FACTOR CRTC1 IN ALZHEIMER DISEASE TRANSGENIC MICE

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