Arterial cerebral infarcts in sickle cell trait and antithrombin III deficiency

Pere Comas; Luis D’Olhaberriague; Merce López; Lluis Soler-Singla; Jordi Pascual; Arantxa Matali; Adolf Pou-Serradell

doi:10.1159/000108836

Arterial cerebral infarcts in sickle cell trait and antithrombin III deficiency

Pere Comas, Luis D’Olhaberriague, Merce López, Lluis Soler-Singla, Jordi Pascual, Arantxa Matali, Adolf Pou-Serradell

Department of Medicine

Research output: Contribution to journal › Article › Research › peer-review

Abstract

Since September 1987, all patients under 46 years admitted to our Hospital with cerebral infarcts (Cl), the cause of which was unknown, have undergone complete hematological screening (partial thromboplastin time; prothrombin ratio; fibrinogen level; lupus anticoagulant activity; fibrinolytic capacity; proteins C and S; antilhrom- bin III; AT-III, and hemoglobin electrophoresis when indicated). In 2 out of 22 patients admitted in 1988, sickle cell trait and AT-III deficiency were the only abnormalities identified. We conclude that complete hematological screening should be carried out in every young patient with Cl when more common causes are not present. © 1991 S. Karger AG, Basel.

Original language	English
Pages (from-to)	168-170
Journal	Cerebrovascular Diseases
Volume	1
Issue number	3
DOIs	https://doi.org/10.1159/000108836
Publication status	Published - 1 Jan 1991

Keywords

Antithrombin III, deficiency of
Ischemic stroke in young
Sickle cell trait

Access to Document

10.1159/000108836

Cite this

@article{c15d528148354226a96c3bf6cdfb989d,

title = "Arterial cerebral infarcts in sickle cell trait and antithrombin III deficiency",

abstract = "Since September 1987, all patients under 46 years admitted to our Hospital with cerebral infarcts (Cl), the cause of which was unknown, have undergone complete hematological screening (partial thromboplastin time; prothrombin ratio; fibrinogen level; lupus anticoagulant activity; fibrinolytic capacity; proteins C and S; antilhrom- bin III; AT-III, and hemoglobin electrophoresis when indicated). In 2 out of 22 patients admitted in 1988, sickle cell trait and AT-III deficiency were the only abnormalities identified. We conclude that complete hematological screening should be carried out in every young patient with Cl when more common causes are not present. {\textcopyright} 1991 S. Karger AG, Basel.",

keywords = "Antithrombin III, deficiency of, Ischemic stroke in young, Sickle cell trait",

author = "Pere Comas and Luis D{\textquoteright}Olhaberriague and Merce L{\'o}pez and Lluis Soler-Singla and Jordi Pascual and Arantxa Matali and Adolf Pou-Serradell",

year = "1991",

month = jan,

day = "1",

doi = "10.1159/000108836",

language = "English",

volume = "1",

pages = "168--170",

number = "3",

}

TY - JOUR

T1 - Arterial cerebral infarcts in sickle cell trait and antithrombin III deficiency

AU - Comas, Pere

AU - D’Olhaberriague, Luis

AU - López, Merce

AU - Soler-Singla, Lluis

AU - Pascual, Jordi

AU - Matali, Arantxa

AU - Pou-Serradell, Adolf

PY - 1991/1/1

Y1 - 1991/1/1

N2 - Since September 1987, all patients under 46 years admitted to our Hospital with cerebral infarcts (Cl), the cause of which was unknown, have undergone complete hematological screening (partial thromboplastin time; prothrombin ratio; fibrinogen level; lupus anticoagulant activity; fibrinolytic capacity; proteins C and S; antilhrom- bin III; AT-III, and hemoglobin electrophoresis when indicated). In 2 out of 22 patients admitted in 1988, sickle cell trait and AT-III deficiency were the only abnormalities identified. We conclude that complete hematological screening should be carried out in every young patient with Cl when more common causes are not present. © 1991 S. Karger AG, Basel.

AB - Since September 1987, all patients under 46 years admitted to our Hospital with cerebral infarcts (Cl), the cause of which was unknown, have undergone complete hematological screening (partial thromboplastin time; prothrombin ratio; fibrinogen level; lupus anticoagulant activity; fibrinolytic capacity; proteins C and S; antilhrom- bin III; AT-III, and hemoglobin electrophoresis when indicated). In 2 out of 22 patients admitted in 1988, sickle cell trait and AT-III deficiency were the only abnormalities identified. We conclude that complete hematological screening should be carried out in every young patient with Cl when more common causes are not present. © 1991 S. Karger AG, Basel.

KW - Antithrombin III, deficiency of

KW - Ischemic stroke in young

KW - Sickle cell trait

U2 - 10.1159/000108836

DO - 10.1159/000108836

M3 - Article

VL - 1

SP - 168

EP - 170

IS - 3

ER -

Arterial cerebral infarcts in sickle cell trait and antithrombin III deficiency

Abstract

Keywords

Access to Document

Fingerprint

Cite this