Polypyrimidine tract binding proteins (PTB) regulate the expression of apoptotic genes and susceptibility to caspase-dependent apoptosis in differentiating cardiomyocytes

Keyphrases

• Cardiomyocytes 100%
• Apoptotic Genes 100%
• PTBP1 100%
• Caspase-dependent Apoptosis 100%
• Anti-apoptotic Proteins 20%
• Caspase-dependent 20%
• Apaf-1 20%
• Ischemia 10%
• Protein Level 10%
• Caspase-3 10%
• Overexpression 10%
• 5′-untranslated Region (5′-UTR) 10%
• Fibroblasts 10%
• Protein-coding Genes 10%
• DNA Fragmentation 10%
• Isoform Expression 10%
• Activity-dependent 10%
• Morphological Abnormalities 10%
• Caspase Activation 10%
• Heart Development 10%
• Caspase Activity 10%
• Developing Heart 10%
• Reporter Gene 10%
• Rat Cardiomyocytes 10%
• Protein Overexpression 10%
• Caspase-independent 10%
• Gene Isoforms 10%
• Protein Silencing 10%
• Embryonic Heart 10%
• Firefly Luciferase 10%
• Myoblast Differentiation 10%

Biochemistry, Genetics and Molecular Biology

• Binding Protein 100%
• Caspase 100%
• Cardiac Muscle Cell 100%
• Polypyrimidine Tract 100%
• Programmed Cell Death 100%
• Gene Expression 30%
• Isoform 10%
• Embryonic Heart 10%
• Caspase 3 10%
• Morphology 10%
• DNA Fragmentation 10%
• Luciferase 10%
• Untranslated Region 10%
• Reporter Gene 10%
• Fibroblast 10%